INTRODUCTION
Headache is one of
the most common of all human physical complaints. Headache is actually a
symptom rather than a disease entity and may indicate organic disease, a stress
response, vasodilation, skeletal muscle tension, or a combination of these
factors.
A primary headache
is one for which no organic cause can be identified. These types of headache
include migraine, tension-type, and cluster headaches.
A secondary
headache is a symptom associated with organic cause, such as a brain tumor or
aneurysm, subarachnoid hemorrhage, stroke, hypertension, meningitis, and head
injury.
(Brunner
& Suddarth’s 2010)
DEFINITION
Headache is probably the
most common type of pain experienced by humans. Headache is a common symptom of
many neurologic conditions and is also a separate disease process.
(Basavanthappa
2009)
ETIOLOGY
1. Sinus
headache
2. Tension-type
headache
3. Migraine
headache
4. Cluster
headache
1)
SINUS
HEADACHE
Sinus
headache are headaches that may feel like an infection in the sinuses
(sinusitis). You may feel pressure around the eyes, cheeks and forehead.
Perhaps your head throbs. But, this pain might actually be caused by a
migraine.
2)
TENSION-TYPE
HEADACHE
Occurs at any age and is
associated with stress. Onset often in adolescence, related to tension or
anxiety. There will be no family history. It is episodic, vary with stress;
duration is variable. The pain is usually bilateral, occurring most often in
the back of the neck. It usually does not interfere with sleep. The headache
occurs intermittently for weeks, months or even years.
3) MIGRAINE HEADACHE
Migraine occurs more often in women than men most
commonly begins between adolescence and at the 40 years. They demonstrate a
strong hereditary pattern, but no specific genetic link has been identified. It
is episodic, tends to occur with stress or life crisis it lasts. Lasts hours to
days. It occurs slowly. Pain becomes severe with one side of head affected more
than other.
4) CLUSTER HEADACHE
Cluster
headache is one of the most severe forms of head pain. No epidemiological
pattern has been identified occurs in early adulthood, precipitated by alcohol
or nitrate use, more common in older men. Episodes clustered together in quick
succession for few days or weeks with remissions that lasts for months. It
lasts a minute to a few hours.
CLINICAL
MENIFESTATIONS
·
SINUS
HEADACHE
o
Bad breath
o
Decreased or absent sense of smell
o
Fever
o
Nasal congestion
o
Stuffy nose
o
Nasal discharge
o
Runny nose
o
Nasal membrane swelling
·
TENSION-TYPE
HEADACHE
o
Dull head pain
o
Pressure around the forehead
o
Tenderness around the forehead and scalp
·
MIGARINE
HEADACHE
o
Aura (sensitivity to light and sound)
o
Vision field defects
o
Confusion
o
Paresthesia
o
Nausea
o
Vomiting
o
Chills
o
Fatigue
o
Irritability
o
Sweating
o
Edema
·
CLUSTER
HEADACHE
o
Flushing
o
Tearing of eyes
o
Nasal stuffiness
o
PATHOPHYSIOLOGY
The
pathophysiology of headache is not fully understood. Some structures of the
head are incapable of sensing pain. The structures that are capable of feeling
pain are skin, muscles, periosteum of the skull, eyes, ears, nasal cavities and
sinuses, meninges, cerebral blood vessels, and cranial nerves with sensory
function. Pain is caused by vasodilation of blood vessels. Serotonin is the
primary neurotransmitter found in the pathways involved in the headache, but
its role is not fully understood.
` When
cerebral blood vessels narrow
Blood
flow is reduced to some areas of the brain
Initial
vasoconstriction is followed by significant vasodilation
Inflammation
of the blood vessels
This
triggers a release of serotonin
Cause
headache
TREATMENT
Medications for the
treatment of headache fall into two broad categories: symptom relief and
prevention.
Ø SYMPTOMATIC TREATMENT
Following drugs are used:
a) Non-narcotic
analgesics (aspirin, acetaminophen and ibuprofen).
b) Analgesics
combinations (butalbital).
c) Muscle
relaxants.
d) Serotonin
receptor agonists (sumatriptan, naratriptan, rizatriptan).
e) Alpha-adrenergic
blockers (ergatramine tartrate).
f) Vasoconstrictors
(isometheptone).
g) Corticosteroids
(dexamethasone).
h) Metodopramide.
Ø PROPHYLACTIC TREATMENT
Includes the following:
a) Tricyclic
antidepressants (doxepin, amitriptyline).
b) Beta-adrenergic
blockers (propranolol, Inderal).
c) Biofeedback.
d) Muscle
relaxation training.
e) Psychotherapy.
f) Calcium
channel blockers (isoptin).
g) Divalp
h) Yoga,
meditation, electric counter stimulation.
i) Corticosteroids
(prednisone).
j) Lithium.
k) Alpha-adrenaline
blockers (used in cluster headache).
l) Serotonin
antagonists.
NURSING CARE PLAN
|
ASSESSMENT |
NURSING DIAGNOSIS |
PLANNING |
NURSING INTERVENTIONS |
EVALUATION |
|
Subjective Data Patient
verbalize that She/he is suffering from severe pain on right side of the head
since 2 to 3 days. Objective Data ·
Blood pressure 130/80mmhg ·
Pulse 88/min ·
Temp 99F ·
Pain scale 7/10 ·
Facial expression. |
Acute
pain Related to stress As evidence by severity of
pain (7/10) Secondary to disease process. |
Short term planning 2hour’s Relieve
pain Calm
& quite environment Relieve
nausea and vomiting Reduce
stress Long term planning Counseling
to coping stress or depression. |
Independent ·
Provide bed ·
Comfortable positioning ·
Attach oxygen if required ·
Secure i/v line ·
Give over the counter drug (paracetamol) ·
Call to doctor ·
Noise free surroundings ·
Encourage adequate rest periods. ·
Increased intake of drink like plenty of water. Dependent ·
Give analgesics as advised by doctor like aspirin ·
Give muscle relaxants as prescribed ·
Antiemetic drugs as order by doctor ·
Give antidepressants as advised Collaborative ·
Send call to psychology for psychotherapy ·
Call to physiotherapy for Muscle relaxation training
·
Advise yoga , meditation for relaxation ·
Counseled the attendants to avoid stressful
environment. |
Short term goal After
2hour’s nursing interventions patient verbalized that his/her pain settled
down and he/she is feeling better. Long term goal After
2weeks follow up patient is feeling better and stress free. |
NURSING CARE PLAN
|
ASSESSMENT |
NURSING DIAGNOSIS |
PLANNING |
INTERVENTION |
EVALUATION |
|
Subjective data Patient
verbalize that She/he is suffering from severe pain on whole head since 3 to
4 days. Objective Data ·
Blood pressure 110/80mmhg ·
Pulse 80/min ·
Temp 98F ·
Pain scale 6/10 |
Disturbed
sleep pattern Related To
anxiety As Evidence By irritability Secondary To Disease process. |
Short term planning 2hour’s Relieve
pain Calm
environment Relieve
nausea and vomiting Reduce
stress. Long term planning Counseling
to coping stress or depression. |
Independent ·
Provide bed ·
Comfortable positioning ·
Attach oxygen if required ·
Monitor vital signs ·
Secure i/v line ·
Give over the counter drug (paracetamol) ·
Call to doctor ·
Keep calm and dim light environment ·
Noise free ·
Advice a regular sleep schedule ·
Advise to improve physical activity in daily routine. ·
Encourage for oral intake of water. Dependent ·
Give analgesics as advised by doctor like aspirin ·
Give muscle relaxants as prescribed ·
Antiemetic drugs as order by doctor ·
Give antidepressants as advised Collaborative ·
Send call to psychology for psychotherapy ·
Call to physiotherapy for Muscle relaxation training
·
Advise yoga , meditation for relaxation ·
Counseled the attendants to avoid stressful
environment. |
Short term goal After
2hour’s nursing interventions patient verbalized that his/her pain settled
down and he/she is feeling better. Long term goal After
2weeks follow up patient is feeling better and stress free. |
NURSING CARE PLAN
|
ASSESSMENT |
NURSING DIAGNOSIS |
PLANNING |
INTERVENTION |
EVALUATION |
|
Subjective data Patient
verbalize that She/he is suffering from severe pain on whole head since 3 to
4 days. Objective Data ·
Blood pressure 130/80mmhg ·
Pulse 88/min ·
Temp 99F ·
Pain scale 7/10 facial. ·
Restlessness. |
Impaired
comfort Related To headache
As Evidence By irritability Secondary To Disease process. |
Short term planning 2hour’s Relieve
pain. Provide
Calm environment. Dim
light but good ventilation. Relieve
anxiety. Reduce
stress. Long term planning Counseling
to coping stress or depression. |
Independent ·
Provide bed ·
Comfortable positioning ·
Attach oxygen if required ·
Secure i/v line ·
Give over the counter drug (paracetamol) ·
Call to doctor ·
Keep calm and dim light environment ·
Encourage adequate rest periods. ·
Counseled to avoid too much use of cellphone and
television. ·
Increased intake of drink like plenty of water. Dependent ·
Give analgesics as advised by doctor like aspirin ·
Antiemetic drugs as order by doctor ·
Give antidepressants as advised Collaborative ·
Send call to psychology for psychotherapy. ·
Call to ophthalmology for vision test. ·
Call to physiotherapy for Muscle relaxation training
·
Advise yoga , meditation for relaxation ·
Counseled the attendants to avoid stressful
environment. |
Short term goal After
2hour’s nursing interventions patient verbalized that his/her pain settled down
and he/she is feeling better. Long term goal After
2weeks follow up patient is feeling better and stress free. |
SUMMARY
Headache
is probably the most common type of pain in humans. Head is often begins in
early morning. The classic migraine attack can be divided into four phases:
prodrome, aura, headache and recovery. Presents in 60% of patients with
migraine headache. Symptoms may occur consistently hours to days before onset
of migraine. Depression, irritability, feeling cold, food cravings, anorexia
and so on noted with headache. There is a period of muscle contraction in the
neck and scalp with associated muscle ache and localized tenderness, exhaustion
and mood changes. The overall goals that patients with headache have reduced or
no pain. Experience increased comfort and decreased anxiety. Demonstrate
understanding of triggering events and treatment strategies. Use positive
coping strategies to deal with chronic pain.
REFERENCES
·
Basavanthappa,
(2009) medical surgical nursing: Headache (2nd edition) page#849.
·
Brunner
& Suddarth’s,(2011) medical surgical nursing: Headache (12th
edition) page#330.
·
Dr.
Inam Danish,(2006) medical diagnosis and management:(8th edition)
page# 650.
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